The Plot Thickens, literally: Pulmonary Embolus and Myocardial Infarction are also caused by Cardiac Arrhythmia.
Of course, you know that recent articles have been published on this site on the link between cardiac arrhythmia and stroke. Now it seems that the plot thickens, literally, since thickened blood is the result from a clotting disorder that is the culprit in the etiology of stroke and embolic diseases. Now we must include pulmonary embolus and its asssociated condition of deep vein thrombosis in our list. Additionally, we must now emphatically state that the reported number one killer of all persons worldwide, heart disease,must now include myocardial infarction, or MI, as it is most commonly referred to, as also being a product of our cardiac killer, SADS (sudden arrhythmic death syndrome. The recent news coverage of the famous athlete, Chris Bosh, and his retirement from the NBA due to his medical issue with phlebitis in the leg and blood clots in the lung, caused me to investigate his case and see if this serial killer is also somehow involved in his condition.
Sure enough, after finding his birth date and place of birth, I was able to deduce his time of birth from his obvious physical characteristics, making it plain to see that he indeed had one of the cardiac killers in his phenotype. The one that he has is called Brugada Syndrome. Please see my previous article called, “Brugada Syndrome surfaces at Sandringham” for further explanation of this disorder.
It appears from the literature that this arrhythmia along with others can cause atrial fibrillation and other cardiac manifestations. Atrial fib, as it is called, is a frequent cause of blood clots and embolization leading to stroke. My previous article explored the possibility that the heart is the culprit in stroke condtions and now it appears that the heart is also the culprit in deep vein thrombosis and even pulmonary emboli….both conditions suffered by Mr Bosh. He was forced to retire because of these, but now it seems that he may not have been treated for his cardiac condition, since the blood thinners he was taking were not adequate to control his disorder, resulting in his inability to pass the physical to play in the NBA. After coming to this realization of my findings, I felt compelled by duty or obligation to publish this and to also provide other article evidence that further corroborates my own conclusions in this matter. I am compelled because this information may save the life of Mr. Bosh…..if , however, we can find a cardiologist willing to do the proper testing and to put him on the appropriate medication for his condition. Brugada Syndrome is treated medically with Quinidine or other similar medications. If these are not helpful then cardiac pacing is then recommended. The article whose findings corroborate mine is linked below.
This article is published by the Journal of the American Medical Association and is entitled, “Blood Clots in Lungs might not always originate in deep veins of the legs and pelvis in trauma patients”. The research was done at Massachusetts General Hospital of the Harvard Medical School and it concluded:
“Based on these data, there is little evidence that pulmonary embolism originates from deep venous thrombosis of peripheral veins”.
They suggest, as my research does also, that clots can form in the leg and in the lung or other areas simultaneously. Perhaps also, the veins and arteries must be considered as really extensions of the heart itself and to be inseparable from the heart, such that the arrhythmia affects not only the heart proper but also its appendages, the veins and arteries. Perhaps contractility not only applies to heart musculature, but also to the contractility of veins and arteries. Indeed, there is a thing called vascular tone which involves the nervous components of the veins and arteries. In my other article, I explored the real situation of decreased oxygen saturation in blood as a trigger for clot formation. Now vascular tone may also be important in the ability of veins and arteries to send blood back to the heart or away from the heart. This is really some important news for the medical community. We will have to rethink our views on the causes of and therefore treatment options for pulmonary embolus and deep vein thrombosis. Since this is now evident, it now becomes highly likely that myocardial infarction is due to arrhythmia and not due to coronary artery disease.
It is already common knowledge that arrhythmia is often the cause of death in a person who has had an MI. The structrual damage to the muscle tissue sets the person up for electrical problems in the heart’s contractile apparatus. However, it again begs an appropriate question, “did the MI cause the arrhythmia or did the arrhythmia cause the MI”. I now proclaim that research needs to now focus its attention on this very real possibility. My research focus will now include those individuals who have had an MI to see if indeed they have the marker or markers for sudden death as Mr. Bosh has shown for pulmonary embolus. Other celebrities who died or had emboli that also had an undiagnosed heart arrhythmia, include Heavy D (Brugada Syndrome)and the retired NBA star, Jerome Kersey.(Wolfe Parkinson White Syndrome). Minister Louis Farrakahn suffered a stroke and has been found to have Long QT Syndrome. From these findings and to potentially save the lives of these survivors of stroke and emboli, it is advised that Mr Bosh be placed on Quinidine and Minister Farrakahn be placed on a beta blocker or other medication as soon as possible to prevent the recurrence of their strokes or emboli. If medications are not indicated then cardiac pacing is suggested.
This implies that myocardial infarction is not a vascular problem in the coronary arteries afterall, in a majority, if not all cases. This finding has immense implications for the cardiology field in general. Hopefully, someone possessing a brain and a soul or conscience will see this evidence as a need to explore this further so that the unnecessary loss of human life can be prevented. It is now time for all persons who have been affected by this condition to demand that the medical community take the appropriate steps to address this problem. Otherwise, we will all have to consider alternatives to the current medical model or practice here in the US and abroad.
1. AAAS(American Archives of Surgery) 19 0ctober 2009. “ Blood Clots in Lungs may not always originate in deep veins of legs and pelvis in trauma patients”..https://www.eurekalert.org/pub_releases/2009-10/mgh-ctf101509.php