The research into the etiology of strokes and sudden cardiac death has been ongoing with the recent discovery of a very important finding. There appears to be a separate cardiac condition from cardiac arrhythmia, that is involved in the predisposition to strokes and clotting disorders.
This condition still involves the heart and vasculature but the arrhythmia component is absent. It seems to involve pure contractility, in that there is a defect in the heart and vessels in the ability to contract effectively to keep the blood flowing in the proper manner. The combination of this problem along with the high protein diet causing sludge in the bloodstream are both co-conspirators in this tendency to form clots. This however does not preclude arrhythmia as a continued culprit in causing strokes, it is just that there is a separate mechanism also at work. So now, there are genetic diseases involving clotting factors in blood causing strokes but also there are cardiac arrhythmia, excess protein and now this contractility defect in the heart musculature, arteries and veins that cause strokes.
This contractility issue also appears to be involved in the tendency to congestive heart failure also, since CHF is usually treated with drugs to increase contractility. In fact my research shows that there can be a combination of decreased contraction power(contractility) and arrhythmia occurring at the same time in one patient. Indeed, several factors mentioned above may occur simultaneously, necessitating a multi drug approach to treatment and or prevention. Blood thinners, anti arrhythmics, and inotropic agents may be considered as needed in select patients. Chris Bosh, now seems to be one of the clients who in addition to blood thinners may need to be placed on an inotropic agent and an antiarrhythmic medication. He also needs to decrease his meat consumption.
Atrial fibrillation, which is very common as a cardiac etiology for stroke, is often a complication of an underlying arrhythmia that we have discussed. Wolfe Parkinson White, Brugada and especially Long QT syndromes are all implicated in the etiology of atrial fibrillation. Hence, treating this condition and not the underlying cause is not adequate treatment.
I have contacted several medical centers regarding my research and no one as of yet has the courage to invite my findings for evaluation. This does not look good. You cannot claim a discovery is false unless you do an honest appraisal of the findings. Refusal to do so implies a desire to suppress that which may prove current models wrong. This is childish and even dangerous. Those of you who discredit my findings outright only prove that you are hiding something and are afraid that I might be correct. You are harming the practice of medicine. If you will not give my findings due just appraisal, you are wasting your time libeling me and my work. I have no respect for your biased opinion. Your are a bigot who refuses to listen to any approach that does not come from the halls of western medicine. Shame on you. You are only harming yourself and the respect for medical practice under the current model.
I will continue to do my work and try to offer it to the public.